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Description: Alok Sharma is a Pathologist of Non Clinical Safety Assessment at Covance Laboratories Inc. Madison, USA. He earned his PhD in Toxicologic Pathology from Department of Comparative Pathobiology at Purdue University. He was member of the Editorial Board of Journals in the area Veterinary Pathology Toxicologic Pathology.
Description: Alok Sharma, BVSc, MVSc, MS, PhD, DACVP, DABT, Covance 8:10 AM–9:00 AM Comparative Anatomy of the Eye and Ocular Structures of Laboratory Animals Steve Sorden, DVM, PhD, Covance 9:00 AM–10:00 AM Physiology of the Eye and Ocular Structures of Laboratory Animals Joshua Bartoe, MS, DVM, Northern Biomedical Research 10:00 AM–10:30 AM Break
Description: Dr. Alok Sharma (Covance Laboratories) reiterated the definitions of adaptive, nonadverse, and adverse responses to emphasize that adaptive responses are not always nonadverse and can become adverse when severe enough as described by Palazzi et al. (2016). The adaptive nature of a pathologic finding must not be used to regard it as nonadverse.
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Description: Alok Sharma, BVSc, MVSc, MS, PhD, Diplomate ACVP, DABT. Dr. Alok Sharma is an Associate Director (Pathology) at Covance Laboratories Inc., Madison WI, USA. He is a diplomate of ACVP and ABT. He completed his residency in Anatomic Pathology and received a PhD in Experimental Toxicologic Pathology from Purdue University.
Description: Table of contents for Toxicologic Pathology, 48, 1, Jan 01, 2020. Access to society journal content varies across our titles. If you have access to a journal via a society or association membership, please browse to your society journal, select an article to view, and follow the instructions in this box.
Description: Educational Courses Pathology for Nonpathologists Course Description. This joint ACT/STP course, held every other year, will be held at the Gaithersburg Marriott Washingtonian Center, Gaithersburg, Maryland. The lectures are given by highly respected pathologists with current, relevant experience in toxicologic pathology.
Description: Putative gain-of-function mutations in leucine-rich repeat kinase 2 (LRRK2), resulting in increased kinase activity and cellular toxicity, are a leading genetic cause of Parkinson’s disease (PD). Hence, there is strong interest in developing LRRK2 kinase inhibitors as a disease-modifying therapy. Published reports that repeat dosing with two LRRK2 kinase inhibitors (GNE-7915 and GNE-0877 ...
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